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n. The research team included collaborators from the MRC Centre for Medical Molecular Virology and the Retrovirus Research Laboratory, MRC University of Glasgow Centre for Virus Research.

This has been characterised by the concentration of populations into larger settlements and the attempted suppression or submergence of endogenous belief systems in favour of world religions or political ideologies.

Galectin-3, a novel endogenous TREM2 ligand, detrimentally regulates inflammatory response in Alzheimer’s disease.. ... Galectin-3, A Novel Endogenous Trem2 Ligand, Regulates Inflammatory Response and Aβ Fibrilation in Alzheimer’s Disease.

Toggle navigation. Go. SCOPe. Domain. Species. Proteins. 1. 1. Please cite: Gough, J., Karplus, K., Hughey, R. and Chothia, C. (2001). "Assignment of Homology to Genome Sequences using a Library of Hidden Markov Models that Represent all Proteins of

InterPro (1). 0.0. 100382807. Retrovirus-related Pol polyprotein LINE-1. None. EMBL (3).

Pathogenic viral and non-pathogenic endogenous nucleic acids are often difficult to distinguish. ... Endogenous RNA is abundant in the cytosol, and viruses have evolved various strategies to mask their most proinflammatory nucleic acid signatures.

Endogenous Nmnat2 is an essential survival factor for maintenance of healthy axons..

Toggle navigation. Go. Acyl carrier protein-like. Superfamily. Families. Domains. 15. 10. 3. 5. 7. 8. Please cite: Gough, J., Karplus, K., Hughey, R. and Chothia, C. (2001). "Assignment of Homology to Genome Sequences using a Library of Hidden

Toggle navigation. Go. SCOPe. Domain. Species. Proteins. 1. 1. Please cite: Gough, J., Karplus, K., Hughey, R. and Chothia, C. (2001). "Assignment of Homology to Genome Sequences using a Library of Hidden Markov Models that Represent all Proteins of

Toggle navigation. Go. Small proteins. Fold. Superfamilies. Families. 1. 1. 4. 4. 19. 62. 1. 1. 1. 2. 1. 1. 1. 9. 1. 19. 1. 3. 1. 3. 1. 1. 1. 2. 1. 1. 1. 4. 3. 3. 1. 19. 1. 9. 1. 6. 1. 2. 1. 2. 1. 2. 1. 1. 1. 4. 1. 1. 1. 1. 1. 2. 1. 2. 1. 1. 1. 1.